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mouse anti s 100β chain  (Santa Cruz Biotechnology)


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    Santa Cruz Biotechnology mouse anti s 100β chain
    Mouse Anti S 100β Chain, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 33 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+s+100%CE%B2+chain/S-100+%CE%B2+chain+Antibody/pmc10857775-138-20-24
    Average 93 stars, based on 33 article reviews
    mouse anti s 100β chain - by Bioz Stars, 2026-09
    93/100 stars

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    other:

    Article Title: The Primary Ciliary Deficits in Cerebellar Bergmann Glia of the Mouse Model of Fragile X Syndrome
    Article Snippet: The following primary antibodies were used in this study: mouse anti-BLBP (1:200; Abcam; ab131137), mouse anti-Sox-2 (1:100; Santa Cruz; sc-365964), mouse anti-S-100β chain (1:100; Santa Cruz; sc-393919), mouse anti-NeuN (1:500; Millipore-Sigma; MAB-377), chicken anti-calbindin (1:400; Encor; CPCA-Calbindin), rabbit anti-Arl13b (1:700; Proteintech; 17711–1-AP), rabbit anti-BLBP (1:300; Abcam; ab32423), rat anti-BrdU (1:200; Novus; NB500–169), and rat anti-BrdU (1:200; Abcam; ab6326).

    Article Title: The Primary Ciliary Deficits in Cerebellar Bergmann Glia of the Mouse Model of Fragile X Syndrome.
    Article Snippet: Primary cilia are non-motile cilia that function as antennae for cells to sense signals.. Deficits of primary cilia cause ciliopathies, leading to the pathogenesis of various developmental disorders; however, the contribution of primary cilia to neurodevelopmental disorders is largely unknown.. Fragile X syndrome (FXS) is a genetically inherited disorder and is the most common known cause of autism spectrum disorders.



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    mouse anti s 100β chain  (Santa Cruz Biotechnology)
    93
    Santa Cruz Biotechnology mouse anti s 100β chain
    Mouse Anti S 100β Chain, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+s+100%CE%B2+chain/S-100+%CE%B2+chain+Antibody/pmc10857775-138-20-24
    Average 93 stars, based on 1 article reviews
    mouse anti s 100β chain - by Bioz Stars, 2026-09
    93/100 stars
    		  Buy from Supplier
    mouse mab s 100β  (Santa Cruz Biotechnology)
    93
    Santa Cruz Biotechnology mouse mab s 100β
    Contribution of RTN astrocytes to breathing disorders in heart failure. (a) RTN relative protein expression levels of FosB, Glial fibrillary acidic protein (GFAP, b) and S100β (c) in Sham rats (n=8) and CHF rats (n=8). (d) measurements of RTN ATP levels in Sham and CHF rats. (e) representative traces of V T and Rf at rest and during central chemoreflex stimulation by hypercapnia (F i CO 2 7%) in CHF rats transfected with an excitatory Designer Receptors Exclusively Activated by Designer Drugs (CHF+DREADD Gq , n=6) and control empty vector (CHF+Ctrl, n=6) following chronic (for 2 weeks) DREADD activation with clozapine-N-oxide (CNO). (f,g) quantification of central chemoreflex sensitivity in CHF+Ctrl and CHF+DREADD Gq rats, (f) the difference in minute ventilation (V E ) in hypercapnia vs. baseline ventilation and (g) the hypercapnic ventilatory response (HCVR). (h) representative traces of ventilation of one CHF+Ctrl and one CHF+DREADD Gq rat. Arrowheads showing apnoeas and hypopneas. (i,j), representative plots of ventilatory variability at rest. (I) Poincare plots showing breath-to-breath T TOT variability. (j) histograms of V T . (k-m) measurements of respiratory instability during resting ventilation. (k), irregularity score (IS), (l), coefficient of variation of V T . (m) histological confirmation of RTN astrocytic viral transfection of the excitatory DREADD (mCherry fluorescence) (scale bar <t>100</t> µm). (n) western blots experiments showing relative GFAP protein levels in the RTN of CHF+Ctrl (n=6) and CHF+DREADD Gq rats (n=5). (o) measurement of RTN ATP levels. (p) Correlation analysis between GFAP expression and ATP levels. (q) apnoea-hypopnea index (AHI) Data show mean ± s.e.m.; Data was analysed using unpaired t-tests (a-d *: p<0.05 vs. Sham; e-q *: p<0.05 vs. CHF+Ctrl). (a-d).
    Mouse Mab S 100β, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+s+100%CE%B2+chain/S-100+%CE%B2+chain+Antibody/pmc09097632-135-6-13
    Average 93 stars, based on 1 article reviews
    mouse mab s 100β - by Bioz Stars, 2026-09
    93/100 stars
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    Contribution of RTN astrocytes to breathing disorders in heart failure. (a) RTN relative protein expression levels of FosB, Glial fibrillary acidic protein (GFAP, b) and S100β (c) in Sham rats (n=8) and CHF rats (n=8). (d) measurements of RTN ATP levels in Sham and CHF rats. (e) representative traces of V T and Rf at rest and during central chemoreflex stimulation by hypercapnia (F i CO 2 7%) in CHF rats transfected with an excitatory Designer Receptors Exclusively Activated by Designer Drugs (CHF+DREADD Gq , n=6) and control empty vector (CHF+Ctrl, n=6) following chronic (for 2 weeks) DREADD activation with clozapine-N-oxide (CNO). (f,g) quantification of central chemoreflex sensitivity in CHF+Ctrl and CHF+DREADD Gq rats, (f) the difference in minute ventilation (V E ) in hypercapnia vs. baseline ventilation and (g) the hypercapnic ventilatory response (HCVR). (h) representative traces of ventilation of one CHF+Ctrl and one CHF+DREADD Gq rat. Arrowheads showing apnoeas and hypopneas. (i,j), representative plots of ventilatory variability at rest. (I) Poincare plots showing breath-to-breath T TOT variability. (j) histograms of V T . (k-m) measurements of respiratory instability during resting ventilation. (k), irregularity score (IS), (l), coefficient of variation of V T . (m) histological confirmation of RTN astrocytic viral transfection of the excitatory DREADD (mCherry fluorescence) (scale bar 100 µm). (n) western blots experiments showing relative GFAP protein levels in the RTN of CHF+Ctrl (n=6) and CHF+DREADD Gq rats (n=5). (o) measurement of RTN ATP levels. (p) Correlation analysis between GFAP expression and ATP levels. (q) apnoea-hypopnea index (AHI) Data show mean ± s.e.m.; Data was analysed using unpaired t-tests (a-d *: p<0.05 vs. Sham; e-q *: p<0.05 vs. CHF+Ctrl). (a-d).

    Journal: eBioMedicine

    Article Title: Medullary astrocytes mediate irregular breathing patterns generation in chronic heart failure through purinergic P2X7 receptor signalling

    doi: 10.1016/j.ebiom.2022.104044

    Figure Lengend Snippet: Contribution of RTN astrocytes to breathing disorders in heart failure. (a) RTN relative protein expression levels of FosB, Glial fibrillary acidic protein (GFAP, b) and S100β (c) in Sham rats (n=8) and CHF rats (n=8). (d) measurements of RTN ATP levels in Sham and CHF rats. (e) representative traces of V T and Rf at rest and during central chemoreflex stimulation by hypercapnia (F i CO 2 7%) in CHF rats transfected with an excitatory Designer Receptors Exclusively Activated by Designer Drugs (CHF+DREADD Gq , n=6) and control empty vector (CHF+Ctrl, n=6) following chronic (for 2 weeks) DREADD activation with clozapine-N-oxide (CNO). (f,g) quantification of central chemoreflex sensitivity in CHF+Ctrl and CHF+DREADD Gq rats, (f) the difference in minute ventilation (V E ) in hypercapnia vs. baseline ventilation and (g) the hypercapnic ventilatory response (HCVR). (h) representative traces of ventilation of one CHF+Ctrl and one CHF+DREADD Gq rat. Arrowheads showing apnoeas and hypopneas. (i,j), representative plots of ventilatory variability at rest. (I) Poincare plots showing breath-to-breath T TOT variability. (j) histograms of V T . (k-m) measurements of respiratory instability during resting ventilation. (k), irregularity score (IS), (l), coefficient of variation of V T . (m) histological confirmation of RTN astrocytic viral transfection of the excitatory DREADD (mCherry fluorescence) (scale bar 100 µm). (n) western blots experiments showing relative GFAP protein levels in the RTN of CHF+Ctrl (n=6) and CHF+DREADD Gq rats (n=5). (o) measurement of RTN ATP levels. (p) Correlation analysis between GFAP expression and ATP levels. (q) apnoea-hypopnea index (AHI) Data show mean ± s.e.m.; Data was analysed using unpaired t-tests (a-d *: p<0.05 vs. Sham; e-q *: p<0.05 vs. CHF+Ctrl). (a-d).

    Article Snippet: Astrocyte density was assessed with a mouse mAb S-100β (1:1000 dilution; cat# sc-393919 Santa Cruz Biotechnology).

    Techniques: Expressing, Transfection, Control, Plasmid Preparation, Activation Assay, Fluorescence, Western Blot

    P2X7r upregulation in RTN astrocytes restores normal breathing patterns in heart failure. (a) representative traces of one CHF+Ctrl and one CHF rat after transfection with AAV5- GFAP-P2X7r-P2A-GFP (CHF+P2X7r) showing tidal volume (V T ) and respiratory frequency (Rf) at rest and during central chemoreflex stimulation with hypercapnia (F i CO 2 7%). (b) quantification of V E at baseline and during central chemoreceptor stimulation with hypercapnia. (c) representative traces of resting ventilation obtained in one CHF+Ctrl rat and one CHF+P2X7r rat. Arrowheads shows breathing disorders. (d,e) representative plots of ventilatory variability at rest in one CHF+Ctrl rat (n=7) and one CHF+P2X7r rat (n=7) . (d) Poincare plots showing breath-to-breath T TOT variability. (e) histograms of V T . (f,h) measurements of respiratory instability during resting ventilation in the 2 groups. (f) irregularity score (IS), (g) coefficient of variation of V T , (h) apnoea-hypopnea index (AHI). (i) visual confirmation of astrocytic viral transfection in the RTN by GFP fluorescence and GFAP immunolabeling (scale bar 100 µm). (j-k), relative protein expression levels of the P2X7r (j) and GFAP (k) in RTN micro-punches. (l) quantification of RTN ATP levels. Data show mean ± s.e.m.; Data was analysed using unpaired t-tests (c-l *: p<0.05 vs. CHF+Ctrl).

    Journal: eBioMedicine

    Article Title: Medullary astrocytes mediate irregular breathing patterns generation in chronic heart failure through purinergic P2X7 receptor signalling

    doi: 10.1016/j.ebiom.2022.104044

    Figure Lengend Snippet: P2X7r upregulation in RTN astrocytes restores normal breathing patterns in heart failure. (a) representative traces of one CHF+Ctrl and one CHF rat after transfection with AAV5- GFAP-P2X7r-P2A-GFP (CHF+P2X7r) showing tidal volume (V T ) and respiratory frequency (Rf) at rest and during central chemoreflex stimulation with hypercapnia (F i CO 2 7%). (b) quantification of V E at baseline and during central chemoreceptor stimulation with hypercapnia. (c) representative traces of resting ventilation obtained in one CHF+Ctrl rat and one CHF+P2X7r rat. Arrowheads shows breathing disorders. (d,e) representative plots of ventilatory variability at rest in one CHF+Ctrl rat (n=7) and one CHF+P2X7r rat (n=7) . (d) Poincare plots showing breath-to-breath T TOT variability. (e) histograms of V T . (f,h) measurements of respiratory instability during resting ventilation in the 2 groups. (f) irregularity score (IS), (g) coefficient of variation of V T , (h) apnoea-hypopnea index (AHI). (i) visual confirmation of astrocytic viral transfection in the RTN by GFP fluorescence and GFAP immunolabeling (scale bar 100 µm). (j-k), relative protein expression levels of the P2X7r (j) and GFAP (k) in RTN micro-punches. (l) quantification of RTN ATP levels. Data show mean ± s.e.m.; Data was analysed using unpaired t-tests (c-l *: p<0.05 vs. CHF+Ctrl).

    Article Snippet: Astrocyte density was assessed with a mouse mAb S-100β (1:1000 dilution; cat# sc-393919 Santa Cruz Biotechnology).

    Techniques: Transfection, Fluorescence, Immunolabeling, Expressing